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In cases where blood vessels malfunction, like in DR, FSP1 may play a role in controlling the growth of extra blood vessels and the formation of tough scar tissue by communicating with growth factors and the cell surface receptors, which in turn affects how the extracellular matrix changes and the series of events that lead to new blood vessel formation

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However, mitochondrial replacement therapy (MRT) and gene therapy represent promising foundational approaches for treating mitochondrial genetic disorders and preventing their transmission to future generations